Arthritis - Shivax® FT & Shivax® Candicil

Psoriasis Arthritis and Rheumatoid Arthritis are two similar pathologies. A basic difference between these ones is that in psoriasis arthritis there are cutaneous symptom too. Anyway, these two pathologies are founded on a hereditary bent. It means it's written on DNA (genetic code). So, the arthritis will show when toxins level is very high in the body.

In fact these Arthritis have a Poligenical nature, as well as a lot of other pathologies, that is the susceptibility to fall ill is written on more genies and not on a single gene. So probably we can find people who have diabetes of the adult, psoriasis cutaneous, seborrheic dermatitis, hypertension etc. in the same family.

The pathology will show only when a person is in a general stress condition. That is the toxins in the body are really too much.

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A little example...

In a car we have a switchboard, where there are a lot of bright lights that can be switched on to indicate that something doesn't work. In the "human machine" these lights are represented by most various symptoms. The lesions of joints and rheumatoid arthritis are like warning lights that come out to highlight that there are too many toxins in our body.

But where do they come from ?

	From an incorrect and unhealthy diet

	From the liquids we drink

	From the air that penetrates our lungs

	From the thoughts that crowd our mind

In the female genital area an excessive Candida proliferation could cause white leaks, (similar to "cottage cheese"), burnings after urinating, and rednesses in the same area.

In male genital area Candida causes penis inflammations, many little, velvety, shining, erythematous spots (red) placed in the penis and characterized by burnings and itches.

Anyway there is always an immunity defense decrease.

Thanks to many international searcher studies (see bibliography) and my experience too, it's obvious that Psoriatic and Rheumatoid Arthritis are in relation with Candida Albicans chronic infection, as well as other disease with psychosomatic components.

Candida Albicans is a fungus we can find in our organism since the birth. In fact when the baby borns he crosses the birth channel, and he gets infected, because this fungus lives in a large quantity in vaginal secretion of pregnant women. From this moment Candida will colonize the little intestine mucous , its real home , but if the colonies are not so much , it will be only a saprophyte , that's is it will be not dangerous. If the immunitary system will get depressed, the number of the colonies will increase.

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An example: imagine to observe a healthy wood, after an heavy rain we will see to be born several fungi, scattered in the underbrush. Then fungi will be present in not excessive quantity. In the case in which we were to observe a tree with umpteen fungi, this will mean that the tree no longer enjoys good health. The same happens in the human organism, with the difference that fungi, that is candida is much smaller.

Now, an external Candida infection show it's possible, through Candida "second houses", such as vaginal infections, burnings at penis level, fungus skin spots, "thrush", foot of athlete and so on. Instead , the 79 toxins produced by Candida are the most silent ones, they are directed towards the central nervous system and to the primary and secondary immunitary system (the intestinal one).

Candida infection lives in all the people who suffer of psychosomatic pathologies. Remember that those toxins can modify your thoughts. Have you ever wish very much Pane (Bread), Pasta, Pizza, Potatoes (called by Dr. ENZO DI MAIO "Four P"), Sweet or Sugar? If yes, that's not your body asking for them but Candida is. It feeds itself of simple carbohydrates (sugar) and when it needs them it sends some toxins to your brain to make you eat. After about fifteen minutes its lunch will be over and what it sends some more toxins to your brain to make you stop eating You will feel your stomach bloated so you will think "but what did I eat? I've just had a small piece of bread or a small piece of cake..."

This that follows, wants to be a popular and informative display, about the existing therapeutic aids, or that we know today. Must not be used for self-medication.

	Interview RAI, Mi Manda RAI 3	Phone Orders and Information

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The list of pathologies where you can find Candida could be very long. Just to remember someone of them:

The smaller ones: gastritis, colitis (either with constipation or diarrhea), several digestives disorders, Depression (see Syndrome from Chronic Hard work),Anxiety, excessively FAT, Anorexia, Insominia, difficult menstruation and before menstrual syndrome, Vaginity or recurrent Cistitis, Balanopostitis (Penis inflammation), Herpes in its various manifestations, Condylom and little Condylom from HPV (Human Papilloma Virus), Headache, alimentary Intolerances, Seborrheic Dermatitis etc.…

Most considerable: Psoriasis, Rheumatoid Arthritis, Psoriatic Arthritis, Vitiligo, Lupus, Crohn's disease, Diabetes of adult, Chronic Depresssion, Gastro - Duodenal Ulcer, Allergic Asthma and not, Arterial Hypertension etc...

Do you think they're very much? the American statistics say Candida chronic infection shoots 70% and 80% of the world-wide population, (see bibliography). In the last 15 - 20 years there was a remarkable increasing of many pathologies named psychosomatics and a lot of more general manifestations due to Candida. This is due to many factors, as: the antibiotics indiscriminate use, central nervous system drugs (anti-depressives, tranquillizers, sleeping pills), cortisones, contraceptive pills, anti-ulcer drugs (cimetidina, etc), massive use of dental amalgam (the dark clogging, contain mercury), the exaggerated use of conserving and colouring, but also of grain products.

It's possible doing many clinical checking as specific antibody search for Candida in blood, in feces, in urine, in vaginal or penis secretion but it can be very useful the use of some studied questionnaires for statistics or for the correlated symptoms.

However in any case shouldn't be eliminated only "the Four P of Dr. DI MAIO MD" ©Copyright 1995. Because just eliminating them, without a correct feeding designed exclusively for that individual according to the Ayurvedic Medicine philosophy and the most current knowledge of Western Medicine, candida after a few days will send neurotransmitters to the brain so that it's not possible to resist and so the individual will end up to overeat of one or all " Four P of Dr. DI MAIO MD" ©Copyright 1995.

The result will be extremely negative. To can bring back the lost balance it will be able make a specific "Personal Protocol" (see at the page

http://www.psoriasi.org/psoriasis/personalprotocol.htm )

To restore the balance it will carry on itself General Approach according to Dr. DI MAIO's Method, that includes both an alimentary discourse and an not alimentary discourse.

Once found the Candida chronic infection (Chronic Candidiasis Syndrome or Candida Related Complex for anglosaxons) the best thing to do is to formulate a personal therapy considering totally the subject, verifying his individual constitution at first and after his pathology.

It will be possible doing an alimentary and not alimentary speech.

PSORIASIS ARTHRITIS and RHEUMATOID ARTHRITIS approach can be planning:

ALIMENTARY

ALIMENTARY on the basis of individual constitution, according to Ayurvedic Medicine philosophy and to the most modern researchs of Occidental Medicine.

Considering eventual alimentary intolerances, ever present, in these cases like to soft and hard wheat, to yeasts, to milk etc. Taking into consideration the severity of chronic infection by Candida, also with Preliminary Test. Thus with a feeding uprising to restore the lost balance.

Consequently General Approach will be studied singularly for each individual, for that moment of life. Dietary Advice are contained in Personal Protocol

The implementation of nutritional therapy so composed is important, both to deprive Candida of its energy source and to bring back in balance the individual.

NOT ALIMENTARY

Be noted that alimentary and not alimentary discourse can be implemented together.

1. Shivax® FT: 1 application in the morning and 1 application in the night. Shivax® FT is 100% natural, is composed of highly refined white Vaseline, lanolin, essential oil of oenothera biennis, essential oil of triticum vulgaris and essential oil of alternifolia melaleuca. Shivax® FT does not contain water, does not contain alcohol, does not contain perfume, does not contain preservatives.

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2. Detoxification : it can be carried out through vegetable purgatives. It is important to start the therapy with a detoxification in order to eliminate the not digested food of the intestine (that could create toxins).

This could be carried ahead with the "Trittico according to DI MAIO"

3. Shivax® Dermocil: imagine to have our car with low battery. We may be able, also on their own to push it, but if we have some friends that help us it will be easier and faster. "Supplements" represent the friends that help us to push the car with low battery and therefore it will be easier and faster re-activating the immune system. Once the individual will be balanced, the individual can mantain his health and his ideal weight with a correct feeding designed specifically for him or her, without the aid of supplements.

	Minerals: Calcium, Magnesium, Iron, Manganese, Zinc and Copper in balanced batching

	Vitamin C: immunomodulating, with accurate batching
	Vitamin D: remodulative of inmune system,with accurate batching
	Vitamin A: reparative, as immunostimulant
	Omega 3 and Omega 6 natural and balanced: powerful antioxidants, be taken three times daily
	Vitamin E: antioxidant, to strengthen the immune system
	Vitamins of group B: as immunostimulant, without yeasts and without Vitamin B12 (Vit. B12 increase Candida)

	All the active ingredients mentioned above are present in high quantities and perfectly balanced in Shivax® Dermocil Take 1 capsule before breakfast, 1 capsule before lunch and 1 capsule before dinner		Phone Orders and Information

4. Physical Exercise : practiced regularly, for several reasons. It allows the elimination of toxins through the sweat and it contributes to regularize the intestinal motility.

5. Meditation: it could be carried out regularly, twenty or thirty minute a day.

6. Alimentary Intolerance : if the Candida Infection is chronic (Chronic Candida Syndrome or CCS) this could be caused by alimentary intolerances, supported by the alterations that Candida creates in the mucous of the small intestine.

7. Partner's Control: It is necessary, because Candida's infection can be located in the genital area too, vulvo-vaginal mucous in women and penis in men. So it's important to do a fresh smear in occasion of a gynecological visit even for the partner. Obviously in this case it will be important to extend to both the therapy.

This article has been written with the scientific collaboration of Dr. Enzo DI MAIO, that from more than 30 years pratices successfully these methods.
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BIBLIOGRAPHY

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Schwartz, RH. Knerr, RJ. Candida esophagitis during treatemnt for adolescent acne vulgaris. Pediatric Infectious disease. 1(5):374, Sep-Oct, 1982.
Jayagopal, S. Cervia, JS. Colitis due to Candida albicans in a patient with AIDS. Clinical Infectios Diseases. 15(3):555, Sep. 1992.
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Nelson, Robert S. Bruni, Hamilton C. Goldstein, Harvey M. Primary gastric candidiasis in uncompromised subjects. Gastrointestinal Endoscopy. 22:2, 92-94, 1982.
Chan, Stephen, PhD, of SUNY College at Brockport, NY. Conversations with.
Discussions with patients that have been treated with antifungal and diet therapy.

Candida and Psoriasis in Dermatological Disease

Skinner, RB. Jr. Rosenberg, W. Noah, PW. Psoriasis of the palms and soles is frequently associated with oropharyngeal Candida albicans. Acta Dermatological Venereol Supplement. 186:149-150, 1994.
M buslau, Menzel I, Holzmann H. Fungal flora of the human faeces in psoriasis and atopic dermatitis. Mycoses. 33:2, 90-4, Feb. 1990.
Soyeur U. Kilic H. Alpan O. Anti-Candida antibody levels in psoriasis vulgaris. Cent. Afr. Journal of Medicaine. 36: 8, 190-2, Aug. 1990.
Baker BS. Powles AV. Malkani AK. Altered call-medicated immunity to group A haemolytic atreptococcal antigens in chronic plaque psoriasis. British Journal of Dermatology. 125: 1, 38-42, Jul 1991.
el-Maghrabi EA. Dixon DM. Burnett JW. Characterization of Candida albicans epidermolytic proteases and their role in yeast-cell adherance to keratinocytes. Clinical Experimental Dermatology. 15: 3, 183-91, May 1990.
Senff H. Bothe C. Busacker J. Reinel D. Studies on the yeast flora in patients suffering from psoriasis capillitii or seborrheic dermatitis of the scalp. Mycoses. 33:1, 29-32, Jan 1990.
Orkin VF. [The characteristics of the clinical picture of candidiasis of the skin and mucous membranes in patients with chronic dermatosis] - Russian. Vrach Delo. 5, 78-80, May 1992.
McKay M. Vulvar dermatoses: common problems in dermatological and gynecological practice. British Journal of Clinical Pract. Sym. Supplement. 71: 5-10, Sep 1990.
Noah PW. The role of microorganisms in psoriasis. Semin Dermatology. 9:4, 269-76, Dec 1990.
Haneke E. Fungal infections of the nail. Semin Dermatology. 10: 1, 41-53, Mar 1991.
Rosenberg, EW. Noah PW. Skinner RB. Microorganisms and psoriasis. Journal of the National Medical Association. 86:4, 305-10, Apr 1994.
Meinhof W. [Intestinal colonization with Candida albicans and its effect on chronic inflammatory dermatoses]-German. Hautarzt. 46:8, 525-7, Aug 1995.
Buslau L. Hanel M. Holzmann H. The significance of yeasts in seborrheic eczemna. Hautarzt. 40(10):611-3, Oct. 1989. - German
Henseler T. [Mucocutaneous candidiasis in patients with skin diseases] - German. Mycoses. 38 Supplement 1:7-13, 1995.
Kemeny L. Ruzicka T. Dobozy A. Michel G. Role of interleukin-8 receptor in skin. International Archives of Allergy and Immunology. 104: 4, 317-22, Aug 1994.
Squiquera L. Galimberti R. Morelli L. Plotkin L. Milicich R. Kowalckzuk A. Leoni J. Antibodies to proteins from Pityrosporum ovale in the sera from patients with psoriasis. Clinical Experimental Dermatology. 19: 4, 289-93, Jul 1994.
Oranje AP. Dzoljic-Danilovic G. Michel MF. Aarsen RS. van Joost, T. [Is juvenile seborrheic dermatitis a candidiasis? Studies of a possible link with microbial infections.] - German Tijdschrift voor Kindergeneeskunde. 55(3):87-92, Jul 1987.

Candida and Diarrhea

Burke, V., Gracey, M. An experimental model of gastrointestinal candidiasis Journal of Medical Microbiology. 13:103-110.
Gupta, T., Ehrinpreis, M. Candida-associated diarrhea in hospitalized patients. Gastroenterology. 98:780-785, 1990.
Danna, P., Urban, C., Bellin, E., Rahal, J. Role of candida in pathogenesis of antibiotic-associated diarrhoea in elderly inpatients. The Lancet. 337:511-514, 1991.
Bishop, R., Barnes, G. Depression of lactase acitivity in the small intestines of infant rabbits by Candida albicans.
Kane, J., Chretien, J., Garagusi, V. Diarrhoea caused by Candida The Lancet. 335-336, 1976. (Immunocompetent).
Garagusi, VF. Chretien, JH. Diarrhoea caused by Candida.(letter) Lancet. 1(7961):697-8, Mar 27, 1976.
Letter in Lancet in response. Enweani IB. Obi CL. Jokpeyibo M. Prevalence of Candida species in Nigerian children with diarrhoea. J.Diarrhoeal Dis Res 12(2):133-5, Jun, 1994.
Gut flora in normal and disordered states. Chemotherapy. 5-15, 1995.
Vogel LC. Antibiotic-induced diarrhea. Orthop Nurs 14(2): 38-41, Mar-Apr, 1995.
Koffi-Akoua G. Ferly-Therizol M. Kouassi-Beugre MT. Konan A. Timite AM. Assi Adou J. Assale G. [Cryptosporidium and candida in pediatric diarrhea in Abidjan.] Bull Soc Pathol Exot Filiales 82(4): 451-7, , 1989.
Ngan PK. Khanh NG. Tuong CV. Quy PP. Anh DN. Thuy HT. Persistent diarrhea in Vietnamese children: a preliminary report. Acta Paediatric Supplement. 381: 124-6, Sep, 1992.
Siregar CD. Sinuhaji AB. Sutanto AH. Spectrum of digestive tract diseases 1985-1987 at the Pediatric Gastroenterology Outpatient Clinic of Dr. Pirngadi General Hospital, Medan. Paediatr Indones. 30(5-6): 133-8, May-Jun, 1990.
Talwar P. Chakrabarti A. Chawla A. Mehta S. Walia BN. Kumar L. Chugh KS. Fungal diarrhoea: association of different fungi and seasonal variation in their incidence. Mycopathologia. 110(2): 101-5, May, 1990.
Omoike IU. Abiodun PO. Upper small intestinal microflora in diarrhea and malnutrition in Nigerian children. Journal of Pediatric Gastroenterolog Nutrition 9(3): 314-21, Oct, 1989.

Immunosuppression

Hirschel B. [AIDS and gastrointestinal tract: a summary for gastroenterologists and surgeons] Schweiz Med Wochenschr. 120(14): 475-84, Apr 7, 1990.
Gage TP. Eagan J. Gagnier M. Diverticulitis complicated by candidal pylephlebitis. South Med. Journal 78(10): 1265-6, Oct, 1985.
Caselli M. Trevisani L. Bighi S. Aleotti A. Balboni PG. Gaiani R. Bovolenta MR. Stabellini G. Dead fecal yeasts and chronic diarrhea. Digestion. 41(3): 142-8, 1988.
Zhen DL. [Analysis of the causative organisms in adult acute infectious diarrhea encountered in the past 12 years]. Chung Hua Nei Ko Tsa Chih 21(9): 540-2, Sep, 1982.
Lorenz A. Grutte FK. Schon E. Muller B. Klimmt G. [Fungal infection of the small bowel mucosa.] Mykosen. 27(10): 506-10, Oct, 1984.

Candida and Antibiotics

[Clinical evaluation of a new oral penem, SY5555, in the pediatric field.] Japanese Journal of Antibiotics. 41-8, Jan, 1995.
Elmer GW. Surawicz CM. McFarland LV. Biotherapeutic agents: A neglected modality for the treatment and prevention of selected intestinal and vaginal infections. Journal of the American Medical Association. 275(11): 870-6, Mar 20, 1996.